[1,2,7] Inspite of the common clinical features, the definitive diagnosis should always be based on clinical, radiological and histological criteria. Surgical treatment is the choice Ponatinib price for condylar OC. The suggested surgical approaches are complete resection of the tumor using condylectomy, condylectomy with reconstruction, or selected tumor removal without condylectomy. The aim of OC treatment should be achieving the acceptable mouth opening, recover facial symmetry, establish facial harmony and occlusion.[1,2,4] CONCLUSION Pain and dysfunction in TMJ region is a diagnostic challenge. TMD are usually diagnosed by thorough patient history, comprehensive clinical examination and radiographic imaging. These pathologies are often initially overlooked, as patients are treated by conventional means.

Imaging techniques are the valuable aid for accurately diagnosing neoplasm like condylar OC. Footnotes Source of Support: Nil Conflict of Interest: None declared.
Adenomatoid odontogenic tumor (AOT) is a rare tumor of epithelial origin comprising 3% of all the odontogenic tumors.[1,2] It was first described by Steensland in 1905. In 1907, AOT was described as pseudo-adenoameloblastoma by Dreibladt.[1] Stafne in 1948 considered AOT as a distinct entity, whereas others believed it to be a variant of ameloblastoma.[3,4] In 1969, Philipsen and Birn declined this thought and suggested the name ��adenomatoid odontogenic tumor��. In 1971, the World Health Organization (WHO) adopted the term ��adenomatoid odontogenic tumor��. Max and Stern, in 2003, coined the name ��adenomatoid odontogenic cyst��.

[3] Various terms like adenoameloblastoma, ameloblastic adenomatoid tumor, adamantinoma, epithelioma adamantinum, and teratomatous odontoma were used before the term AOT.[2,3] AOT is a benign, painless, noninvasive, and slow-growing tumor that does not infiltrate the bone.[5] Clinically, it is often misdiagnosed as an odontogenic cyst. The tumor appears as an intraoral-extraoral swelling in the maxilla and is sometimes referred to as ��two-third tumor�� because it occurs in the maxilla in about two-third cases, about two-third cases arise in young females, two-third cases are associated with an unerupted tooth, and two-third affected teeth are canines.[5,6] The origin of the AOT is controversial. It is thought to arise from odontogenic epithelium because it occurs in the tooth-bearing areas of the jaws, is often associated with the impacted tooth, and has various components of GSK-3 the enamel organ, dental lamina, reduced enamel epithelium, and/or their remnants.